GAD-65/67 Polyclonal Antibody, 100ul Site-directed Mutagenesis and activates c-Jun N-terminal kinase

and activates c-Jun N-terminal kinase (JNK)/stress-activated protein kinase (SAPK) during transient expression in COS and 293 cells

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MIM 600075) and a set of TBP-associated factors (TAFs)

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A pseudogene exists on chromosome 18

GAD-65/67 Polyclonal Antibody, 100ul Site-directed Mutagenesis and activates c-Jun N-terminal kinaseGAD1 encodes one of several forms of glutamic acid decarboxylase, identified as a major autoantigen in insulin dependent diabetes. Glutamate decarboxylase 1 is responsible for catalyzing the production of gamma aminobutyric acid from L glutamic acid. A pathogenic role for this enzyme has been identified in the human pancreas since it has been identified as an autoantigen and an autoreactive T cell target in insulin dependent diabetes. This gene may