DYH5 Rabbit Polyclonal Antibody, 20ul Immune Repertoire Sequencing Mutations in MYL3 have been

Mutations in MYL3 have been identified as a cause of mid-left ventricular chamber type hypertrophic cardiomyopathy

The binding and regulatory activities of this protein have been demonstrated in the regulation of a variety of genes including lactoferrin

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Disruption of the TGF-beta/SMAD pathway has been implicated in a variety of human cancers

and in resulting in poor coordination of speech and body movements

DYH5 Rabbit Polyclonal Antibody, 20ul Immune Repertoire Sequencing Mutations in MYL3 have beenThis gene encodes a dynein protein which is part of a microtubule associated motor protein complex consisting of heavy light and intermediate chains. This protein is an axonemal heavy chain dynein. It functions as a force generating protein with ATPase activity whereby the release of ADP is thought to produce the force producing power stroke. Mutations in this gene cause primary ciliary dyskinesia type 3 as well as Kartagener syndrome which are both