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943 NG/EDA is triggered by the acidic and edaravone-induced high levels of GSH microenvironment, enabling selective and sustained release of edaravone at the site of ischemic injury to enhance its neuroprotective effects

Malaisrie SC, Hofstetter WL, Correa AM, Ajani JA, Komaki RR, Rice DC, Vaporciyan AA, Walsh GL, Roth JA, SG||B |

Based on the p53 regulatory network, small molecule modulators or gene editing tools can be designed, and their therapeutic effects on cartilage protection and inflammation alleviation can be validated in OA animal models