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The opposite directions of the diet and treatment effects are also exemplified by the representative genes (Figure 4D) for processes relevant to the pathogenesis of atherosclerosis, such as leukocyte recruitment ( I L-6, IL-1 receptor antagonist [ I L-1RN], chemokine [C-C motif] ligand 2 [CCL2], leukocyte rolling, adhesion and extravasation (SELE, VCAM-1), cholesterol metabolism and lipid-mediated signaling (ATP-binding cassette transporter 1 [ABCA 1], prostaglandin I2 synthase [PTGIS], extracellular matrix protein turnover [MMP-3 and MMP-13], and plaque hemorrhage [CD163])

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Moreover, a study ( in situ effects on intestinal metabolites and the intestinal mucosal barrier, consequently working through the immune and endocrine pathways to regulate bone mass