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In the oral cavity, a unique feature to be handled by the host defence is that continuously replaced bacteria may obtain a firm anchorage on the nonshedding tooth surface and will thereby remain in close contact with the soft tissues surrounding the tooth for a long time and evoke inflammation (1, 2)

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Preclinical studies demonstrate that DSIP can alter pain threshold responses, with observed effects that are not reversed by opioid receptor antagonists such as naloxone, suggesting involvement of non-opioid or indirect neuromodulatory pathways [7]

Margiana et al., 2022), and possess multilineage differentiation potential

A summary of the potential perioperative improvements suggested can be found in Table 3

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